EFM Exam Dumps - NCC C-EFM Questions and Answers

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

According to NCC, AWHONN, and evidence-based documentation standards, clinicians must document:

Baseline

Variability

Accelerations

Decelerations (type, depth, duration, timing)

Uterine activity

This fulfills the NICHD 3-tier system and legal documentation expectations.

Why the incorrect answers are wrong:

B. "Normal/abnormal" → vague, not an acceptable documentation standard.

C. Category alone → insufficient; categories must be supported by the components.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

Artifact on fetal monitoring:

Appears erratic, disorganized, and without physiologic pattern

Shows random amplitude changes

Often correlates with maternal movement, monitor displacement, or poor signal

Lacks cyclical, repetitive characteristics seen in true dysrhythmias

Fetal dysrhythmias, by contrast:

Have repetitive, patterned, predictable rhythm disturbances

May show uniform premature beats, bigeminy, or sudden rate shifts

Therefore, varied and disorganized = artifact.

Comprehensive and Detailed Explanation From Exact Extract (NCC-Recommended Sources Only)

The fetal heart rate (FHR) tracing shown demonstrates a baseline approximately 135–145 bpm with fluctuations of 6–25 bpm, a hallmark of moderate variability. Moderate variability is defined in all NCC-endorsed resources as the normal amplitude range of 6–25 bpm around the fetal baseline.

According to the AWHONN Fetal Heart Monitoring Principles & Practices (2022–2024), moderate variability is considered the single most reliable indicator of adequate fetal oxygenation and intact neurologic pathways, specifically reflecting well-functioning sympathetic and parasympathetic interplay.

The NICHD/NCC standardized definitions included in the NCC C-EFM Candidate Guide state:

Minimal variability: amplitude range ≤ 5 bpm

Moderate variability: amplitude range 6–25 bpm

Marked variability: amplitude > 25 bpm

Sinusoidal pattern: smooth, undulating waveform, 3–5 cycles per minute, equal amplitude, absent beat-to-beat variability

The tracing provided does not show the repetitive, smooth, wave-like pattern of a sinusoidal rhythm; nor does it show flattening associated with minimal variability. Instead, it includes continuous beat-to-beat fluctuation within the moderate range, without periods of absent or minimal variability.

Menihan’s Electronic Fetal Monitoring (5th ed.) and Simpson & Creehan’s Perinatal Nursing (5th ed.) both emphasize that moderate variability is:

A reassuring feature

Indicative of adequate fetal CNS oxygenation

Expected in a reactive, well-oxygenated fetus

A key criterion for Category I classification

Additionally, Miller’s EFM Pocket Guide reiterates that variability between 6–25 bpm is considered the normal (moderate) fetal autonomic response and is not a sinusoidal pattern, which has a fixed amplitude and frequency.

Therefore, based on NCC-standard definitions and the observed amplitude, the correct interpretation is moderate variability.

References (No URLs):

AWHONN Fetal Heart Monitoring Principles & Practices; NCC C-EFM Candidate Guide 2025; Simpson & Creehan Perinatal Nursing; Menihan Electronic Fetal Monitoring; Miller’s Pocket Guide to Fetal Monitoring; Creasy & Resnik Maternal-Fetal Medicine.

Comprehensive and Detailed Explanation From Exact Extract (No URLs)

For fetuses before 32–34 weeks, the National Certification Corporation (NCC) follows the physiologic standards established by AWHONN, Simpson & Creehan, Menihan, and Creasy & Resnik, which emphasize that preterm fetuses have less mature autonomic nervous system development, resulting in smaller and shorter accelerations.

According to the NCC C-EFM Exam Content Outline (Pattern Recognition & Intervention) and the AWHONN Fetal Heart Monitoring Principles (2022–2024):

Preterm fetuses (<32 weeks) normally demonstrate 10 bpm × 10 sec accelerations.

By approximately 32–34 weeks, accelerations may begin transitioning toward 15×15, but the accepted standard for documentation at 34 weeks remains 10×10, unless clearly meeting 15×15 criteria.

NCC emphasizes using gestational-age–appropriate criteria for documenting accelerations, because autonomic reactivity increases gradually and is not fully comparable to term until after 32–34 weeks.

Menihan’s Electronic Fetal Monitoring also states that preterm fetuses “should be evaluated with the 10×10 rule until it is clear that the fetus is demonstrating mature 15×15 acceleratory capacity.”

Simpson & Creehan reinforce this point, noting that accelerations in late preterm gestations “may not consistently reach 15 bpm for 15 seconds, and thus 10×10 remains the appropriate designation.”

Since the patient is 34 weeks, the fetus is late-preterm and may not reliably meet the full 15×15 criteria; therefore, the correct documentation standard remains 10×10.

Thus, accelerations should be charted as:

“Present 10×10.”

References

NCC C-EFM Candidate Guide 2025 – Content Domain: Pattern Recognition and Intervention

AWHONN Fetal Heart Monitoring Principles & Practices, 2022–2024

Menihan: Electronic Fetal Monitoring: Concepts and Applications

Simpson & Creehan: Perinatal Nursing

Miller: Fetal Monitoring Pocket Guide

Creasy & Resnik: Maternal–Fetal Medicine

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

NCC emphasizes that only objective acid-base assessment can diagnose intrapartum asphyxia. This is accomplished with cord arterial blood gas analysis showing:

pH < 7.0–7.1

Base deficit ≥ 12 mmol/L

Elevated PCO₂

FHR patterns suggest risk, but do not diagnose asphyxia.

Apgar scores, especially at 1 minute, do not correlate reliably with acidemia.

Thus, cord gas analysis is the correct determinant.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

This tracing demonstrates:

A sudden prolonged deceleration following epidural placement

Minimal variability during the deceleration

Event occurring within 15 minutes of epidural

NCC, AWHONN, and Menihan emphasize that maternal hypotension is the most common complication immediately following epidural analgesia. Hypotension leads to:

Reduced uteroplacental perfusion

Fetal bradycardia or prolonged decelerations

Decreased variability during the deceleration

Typical fetal response to maternal hypotension:

Late-like or prolonged deceleration with weakening variability, exactly like the strip shown.

Therefore, the FIRST and most critical step is to check maternal blood pressure.

Other options:

B. Continue to monitor — unsafe when a prolonged deceleration is present.

C. Cervical exam — not indicated; the fetal tracing deterioration is temporally linked to epidural placement.

Thus, the correct action is A. Check maternal blood pressure.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

Vagal stimulation is part of the parasympathetic nervous system, which causes:

Slowing of the fetal heart rate (FHR)

Rapid but temporary changes in HR

Seen with head compression, scalp stimulation, or fetal movement

NICHD/NCC physiology explains:

Vagus nerve activation → acetylcholine release → slowed SA node firing → decrease in FHR

This mechanism is responsible for early decelerations during labor due to head compression.

Why the incorrect answers are wrong:

B. Increased cardiac contractility → sympathetic effect, not vagal.

C. Increased fetal blood pressure → also a sympathetic effect.

Correct answer: A. Decreased fetal heart rate

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

Post-dates surveillance focuses on:

Amniotic fluid volume (AFI or deepest vertical pocket)

This is the most sensitive parameter of placental function

Oligohydramnios is strongly associated with post-maturity and perinatal morbidity

NCC and AWHONN emphasize amniotic fluid as the primary parameter for fetal well-being in post-term surveillance.

Why the incorrect answers are wrong:

B. Fetal weight → inaccurate and not used for surveillance decisions.

C. Placental calcification → poor predictor of fetal outcome and not used for management.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

As gestation advances:

Vagal (parasympathetic) control increases,

Sympathetic dominance decreases,

Resulting in a lower baseline heart rate.

NCC physiology teaching:

“Baseline FHR decreases with advancing gestational age due to maturation and increasing parasympathetic tone.”

Why the others are incorrect:

Catecholamines increase heart rate, not decrease it.

Intrinsic ventricular rate does not change significantly with gestational age.

Thus, the correct physiologic factor is increased parasympathetic tone.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

The tracing shows tachysystole (more than 5 contractions in 10 minutes) with minimal variability and recurrent decelerations consistent with uteroplacental insufficiency caused by excessive uterine activity.

Dinoprostone (Cervidil) is a uterotonic prostaglandin, and one of its known complications is uterine tachysystole with Category II or III fetal heart rate patterns.

NCC/AWHONN guidance for tachysystole caused by prostaglandins:

FIRST intervention: Remove the dinoprostone insert.

Reposition the patient (already done).

IV fluid bolus (already done).

Consider terbutaline only if tachysystole persists after removal of the agent.

Since maternal repositioning and IV fluids have already failed, the next step is to remove the cervical ripening agent.

Why other answers are incorrect:

A. Continue to observe — Never acceptable with tachysystole + fetal intolerance.

B. Terbutaline — May be used after prostaglandin removal, not before.

Thus, the correct answer is C. Remove the dinoprostone insert.